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It is initiated by changes in intracellular and extracellular lipid peroxidation levels, the balance in GSH oxidation and reduction, and intracellular iron metabolism abnormalities, which promote CNS damage and neuronal death [130]

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Acidic residues at the active sites of CD38 and ADP-ribosylt cyclase determine nicotinic acid adenine dinucleotide phosphate (NAADP) synthesis and hydrolysis activities