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A significant proportion of moderate or partially deficient patients (Class 3) still developed severe hyperbilirubinemia that require prompt phototherapy intervention (14), showing that higher residual enzyme activity level does not confer more protection against hyperbilirubinemia

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Notably, a study in Culex pipiens demonstrated that GSTD1 (CpGSTD1) directly metabolizes permethrin as a substrate [81]

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